cell culture l6 skeletal muscle cells (ATCC)
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cell culture l6 skeletal muscle cells
Cell Culture L6 Skeletal Muscle Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 220 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cell+culture+l6+skeletal+muscle+cells/Primary+Skeletal+Muscle+Cells/pm39322038-49-0-8
Average 95 stars, based on 220 article reviews
Cell Culture L6 Skeletal Muscle Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 220 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cell+culture+l6+skeletal+muscle+cells/Primary+Skeletal+Muscle+Cells/pm39322038-49-0-8
Average 95 stars, based on 220 article reviews
cell culture l6 skeletal muscle cells - by Bioz Stars,
2026-09
95/100 stars
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Cell Culture:Article Title: Metabolic perturbations associated with hIAPP-induced insulin resistance in skeletal muscles: Implications to the development of type 2 diabetes. Article Snippet: The human islet amyloid polypeptide (hIAPP) tends to misfold and selfassemble to form amyloid fibrils, which has been associated with the loss of function and viability of pancreatic β-cells in type 2 diabetes mellitus (T2DM).. The role of hIAPP in the development of insulin resistance (a hallmark of T2DM) in skeletal muscles – the major sites for glucose utilization – needs further investigation.. Even though, insulin-resistant conditions have been known to stimulate hIAPP aggregation, the events that lead to the development of insulin resistance due to hIAPP aggregation in skeletal muscles remain unidentified. Modification:Article Title: Metabolic perturbations associated with hIAPP-induced insulin resistance in skeletal muscles: Implications to the development of type 2 diabetes. Article Snippet: The human islet amyloid polypeptide (hIAPP) tends to misfold and selfassemble to form amyloid fibrils, which has been associated with the loss of function and viability of pancreatic β-cells in type 2 diabetes mellitus (T2DM).. The role of hIAPP in the development of insulin resistance (a hallmark of T2DM) in skeletal muscles – the major sites for glucose utilization – needs further investigation.. Even though, insulin-resistant conditions have been known to stimulate hIAPP aggregation, the events that lead to the development of insulin resistance due to hIAPP aggregation in skeletal muscles remain unidentified. |
![Effects of 5’-N-ethylcarboxamidoadenosine (NECA), 2-(4-[2-carboxyethyl]-phenethylamino) adenosine-52-nethyluronamide (CGS21680), and 8-[4-[4-(4-chlorophenzyl) piperazide-1-sulfonyl) phenyl]]-1-propylxanthine (PSB 603) on interleukin 6 (IL-6) messenger RNA (mRNA) gene expression in rat <t>L6</t> <t>skeletal</t> muscle myotubes using charcoal serum rat L6 skeletal muscle myotubes (70-90% confluent) were serum starved for 7 days and then stimulated for 1 hour IL-6 mRNA levels were measured relative to TATA-BOX using real-time quantitative polymerase chain reaction; stimulation was performed with vehicle (0.1% dimethyl sulphoxide), NECA (10 μM), PSB 603 (100 nM and 1 μM), CGS21680 (100 nM), and forskolin (100 nM), data were represented as means ± standard error of mean of at least three independent experimental groups *denotes p<0.05, **denotes p<0.01 and ***denotes p<0.001, data were analysed using a one-way ANOVA test followed by a Tukey test](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_7993/pmc07227993/pmc07227993__TJPS-14-19-g3.jpg)
![Effects of 5’-N-ethylcarboxamidoadenosine (NECA), 2-(4-[2-carboxyethyl]-phenethylamino) adenosine-52-nethyluronamide (CGS21680) and 8-[4-[4-(4-chlorophenzyl) piperazide-1-sulfonyl) phenyl]]-1-propylxanthine (PSB 603) on interleukin 6 (IL-6) messenger RNA (mRNA) gene expression in rat L6 skeletal muscle myotubes using charcoal serum, rat L6 skeletal muscle myotubes were stimulated for the indicated time from 1 hour to 24 hours and interleukin-6 mRNA levels, relative to TATA-BOX, was measured by quantitative real time polymerase chain reaction; stimulation was performed with vehicle (0.1% dimethyl sulphoxide), NECA (100 nM and 10 μM), PSB 603 (10 μM), CGS21680 (100 nM) and forskolin (1 μM) (a) Stimulation was performed up to 1 hour (b) Stimulation was performed for up to 3 hours (c) Stimulation was performed for up to 24 hours, data were represented as means ± standard error of mean of three independent experimental group (n=3; *denotes p<0.05, **denotes p<0.01 and ***denotes p<0.001), data were analyzed using one way ANOVA test followed by Tukey test](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_7993/pmc07227993/pmc07227993__TJPS-14-19-g4.jpg)